Liquid ohne e Zigarette dampfen?

Sonst kann bei E-Zigaretten ja nichts in Frage kommen!

Einfach mal die Tatsachen zur Kenntnis nehmen:

"NICOTINE IS HIGHLY ADDICTIVE and, along with its metabolites, can cause cancer and affect neuronal development in adolescents irrespective of its source"

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4683316/

"E-CIG DEPENDENCE appears to vary by product characteristics and liquid nicotine concentration, and it may increase over time."

https://pubmed.ncbi.nlm.nih.gov/25332459/

"Patterns of nicotine dependence were comparable across those who had used a combustible cigarette and/or e-cigarette in the past 30 days, with 91.4% of combustible cigarette users, 80.7% of disposable e-cigarette users, 83.1% of pod-based e-cigarette users, and 82.5% of mods/other E-CIGARETTE USERS SHOWING SIGNS OF NICOTINE DEPENDENCE"

https://pubmed.ncbi.nlm.nih.gov/35627381/

"Nicotine is a psychoactive ingredient in tobacco that significantly contributes to the harmful tobacco smoking habit. NICOTINE DEPENDENCE IS MORE PREVALENT THAN DEPENDENCE ON ANY OTHER SUBSTANCE."

https://pubmed.ncbi.nlm.nih.gov/35017179/

"A substantial proportion of U.S. adolescent JUUL USERS REPORTED SYMPTOMS OF NICOTINE DEPENDENCE, which is greater for those who vape more frequently and use other substances."

https://pubmed.ncbi.nlm.nih.gov/34416679/

"THE ADDICTION-RELEVANT MOLECULAR, CELLULAR, AND BEHAVIORAL ACTIONS OF NICOTINE are derived from its stimulatory effects on neuronal nicotinic acetylcholine receptors (nAChRs) in the central nervous system."

https://pubmed.ncbi.nlm.nih.gov/33742685/

"Owing to the relatively mild euphorigenic properties of nicotine, the contribution of the reward-enhancing effects to maintaining dependence is hypothesized to be GREATER FOR NICOTINE THAN OTHER DRUGS OF ABUSE"

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2607327/

"Here, we review the molecular, cellular, and circuit-level mechanisms through which nicotine elicits reward and aversion and the adaptations in these processes that DRIVE THE DEVELOPMENT OF NICOTINE DEPENDENCE."

https://pubmed.ncbi.nlm.nih.gov/35017179/

"The chain of cause and EFFECT OF NICOTINE ADDICTION starts with the interaction of this tobacco alkaloid with nicotinic acetylcholine receptors (nAChRs). This interaction leads to activation of reward centers in the CNS, including the mesoaccumbens DA system, which ultimately leads to behavioral reinforcement and addiction."

https://pubmed.ncbi.nlm.nih.gov/12436424/

"USE OF E-CIGARETTES by U.S. young adults, most of which is not intended to help reduce smoking, is related to more rather than less frequent and intensive cigarette smoking."

https://pubmed.ncbi.nlm.nih.gov/30885517/

"The adaptive changes of nicotinic acetylcholine receptors produced by repeated exposure to nicotine play a crucial role in the establishment of dependence. [...] Endogenous enkephalins acting on mu-opioid receptors also play a role in the development of PHYSICAL DEPENDENCE TO NICOTINE."

https://pubmed.ncbi.nlm.nih.gov/20170672/

Wer diese Fakten leugnen will, der sollte schon mehr vorzubringen haben als die absurde Unterstellung, die Studienautoren könnten nicht zwischen Tabakrauch und Nikotin unterscheiden.